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Shaheen Bibi

Graduate Student


Contact:

Email: shaheen110@ufl.edu
CV

     

     

About

I am a PhD student in Plant Pathology. I completed my masters in virology and immunology from National University of Science and Technology, Pakistan working on evolution of Begomoviruses. I was awarded the prestigious Fulbright Fellowship for 5 years to accomplish my Ph.D. at University of Florida. I was previously working on herbicide resistance under supervision of Todd Gaines at University of Florida. And was later transferred to University of Florida. At UF, I am advised by Dr. Jeffrey Jones. My major focus of research is working on Xanthomonas, causal agent of bacterial spot disease of tomato and pepper. Outside of School, I work as chair to outreach committee at North Florida Fulbright chapter. I also volunteer at UF International students’ speaker’s bureau. I enjoy travelling, seeing natural landscapes.

  • Research

    My research investigates about the host-parasite interaction bacterial leaf spot disease of tomato and pepper caused by Xanthomonas species. Major focus of my study is to explore the virulence mechanisms in xanthomonads in the hosts. I combine techniques from bioinformatics, biotechnology and molecular biology to study the genes involved in virulence and elicitation of hypersensitive response. I also work in collaboration with University of Auburn with Neha Potnis on project to determine the evolution of resistant genes in Xanthomonas Perforans.

  • Publications

    Khan, A. A., El-Sayed, A., Akbar, A., Mangravita-Novo, A., Bibi, S., Afzal, Z., ... & Ali, G. S. (2017). A highly efficient ligation-independent cloning system for CRISPR/Cas9 based genome editing in plants. Plant methods13(1), 86.

    Bibi,S., El-Sayed, A., Huguet-Tapia, J.C., & Ali, G.S.(2019). RNA-Seq Analysis of Phytothora Parasitica Transcriptome after treatment with silver Nano particles. (In progress of submission)

    Naveed, Z, N., Bibi, S., & Ali, G.S., (2018). The Phytophthora RXLR effector Avrblb2 modulates plant immunity by interfering with Ca2+ signaling pathway. (Accepted).